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Reference

Chemistry & identity

The structural family SR-17018 belongs to, how tightly it binds opioid receptors, and the physical properties that constrain how it can be used.

Last reviewed: 2026-05-05Editorial methodology

What kind of compound it is

Its chemical family

The orphine family includes several research compounds (SR-14968, SR-15098, SR-15099, SR-16435) and the illicit-market opioid brorphine. CFSRE alerts have documented an expanding number of analogs detected in the unregulated drug supply[3].

Scientific deep diveMembers of the substituted piperidine benzimidazolone class

The shared scaffold is a piperidine ring substituted at two positions: one bearing a benzimidazolone ring system, the other a benzyl-type fragment. Variation across the family occurs at the benzimidazolone ring (chlorination patterns, propionitrile substitution) and at the second benzyl fragment.

  • SR-14968 — more potent SR-17018 analog from the Bohn lab
  • SR-15098, SR-15099, SR-16435 — research compounds, less characterized
  • Brorphine — encountered in illicit market; implicated in fatal overdoses
  • Chlorphine, cychlorphine, spirochlorphine, spirobrorphine — additional analogs identified by forensic labs

In some forensic alerts, SR-17018 has been reported under the name 5,6-dichloro desmethylchlorphine.

How it binds receptors

"Binding" is how tightly a drug sticks to its target receptor. Lower numbers (in nanomolar, nM) mean tighter binding. SR-17018 binds the μ-opioid receptor (MOR) tightly, the κ-opioid receptor moderately, and barely touches the δ-opioid receptor.

ReceptorKiRelative affinity
μ-opioid receptor (MOR)~11 nMHigh
κ-opioid receptor (KOR)~68 nMModerate
δ-opioid receptor (DOR)>10,000 nMNegligible

How it behaves in cells

In test-tube assays of cells expressing the μ-opioid receptor, SR-17018 strongly activates G-protein signaling but barely recruits β-arrestin2. The ratio between these two is what drove its original characterization as a "highly biased" agonist.

Scientific deep diveIn-vitro functional activity at MOR
AssayEC50Emax
GTPγS binding (G-protein activation)~97–193 nM~72–75%
cAMP accumulation (G-protein signaling)~76 nM~105%
β-arrestin2 recruitment>10,000 nM~10%

The very large gap between G-protein and β-arrestin EC50 values is the basis for SR-17018's original characterization as a "highly biased" MOR agonist[1]. This characterization has since been challenged — see Mechanism of action.

Physical properties

SR-17018 is practically insoluble in water and propylene glycol. It dissolves reasonably well in organic solvents. These properties effectively limit it to oral administration — it cannot be efficiently insufflated, smoked, or injected.

  • Practically insoluble in water and propylene glycol
  • Soluble in DMSO and PEG300
  • Soluble in 10:10:80 DMSO / Tween-80 / water mixtures (community-formulated oral suspensions)

Sources cited on this page

  1. [1]Schmid CL, Kennedy NM, Ross NC, Lovell KM, Yue Z, Morgenweck J, Cameron MD, Bannister TD, Bohn LM. Bias factor and therapeutic window correlate to predict safer opioid analgesics · Cell, 171(5):1165–1175.e13 (2017) doi.org/10.1016/j.cell.2017.10.035
  2. [2]SR-17018 — Wikipedia · Wikipedia en.wikipedia.org/wiki/SR-17018Useful index of primary literature
  3. [3]Public Alerts on Novel Synthetic Opioids — orphine analog series · Center for Forensic Science Research and Education (CFSRE) www.cfsre.org/nps-discovery/public-alerts